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MOTS-c

$219$43,900

99% Pure | USA Made | Multi Dose

MOTS-c is a mitochondria-derived peptide that regulates metabolic homeostasis, supports exercise capacity, and promotes healthy aging at the cellular level.

Dose

$219

1

Pairs well with

Bacteriostatic Water

Required for reconstitution

$12

Note: Peptides are not ready to use. Must purchase BAC water for reconstitution.

Key Benefits

What makes MOTS-c exceptional

Every treatment in our catalog is physician-reviewed and pharmacy-compounded. Here's why MOTS-c stands out.

First peptide to demonstrate mitochondria-to-nucleus retrograde signaling

Overview

What is MOTS-c?

MOTS-c (Mitochondrial Open Reading Frame of the Twelve S rRNA type-c) is a recently discovered peptide encoded within the mitochondrial genome. It acts as a cellular energy regulator, activating the AMPK pathway to optimize glucose metabolism, enhance exercise capacity, and promote metabolic flexibility. It represents a breakthrough in mitochondrial medicine and metabolic optimization.

10mg weekly · Subcutaneous injection

Administered under physician guidance

Why choose MOTS-c?

MOTS-c targets aging at its root — mitochondrial function. Patients choose it for:

Enhanced exercise performance and endurance capacity

Improved insulin sensitivity and glucose utilization

Cellular-level anti-aging through mitochondrial optimization

Fat metabolism enhancement via AMPK pathway activation

Protection against age-related metabolic decline

The Difference

What sets MOTS-c apart

MOTS-c is the first mitochondria-derived peptide shown to regulate nuclear gene expression — a breakthrough concept called ‘retrograde signaling.’ It literally allows your mitochondria to communicate with and reprogram your cell nuclei for better metabolic function. No other peptide works at this fundamental cellular level.

Clinical Evidence

Backed by research

Peer-reviewed studies supporting MOTS-c treatment protocols.

2021Cell Metabolism

MOTS-c and Exercise Endurance in Aging

MOTS-c treatment improved physical capacity and reversed age-dependent insulin resistance in models.

Ready to start your treatment?

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